[Latin Name] Rhodiola Rosea

[Plant Source] China

[Specifications] Salidrosides:1%-5%

Rosavin:3% HPLC

[Appearance] Brown fine powder

[Plant Part Used] Root

[Particle size] 80 Mesh

[Loss on drying] ≤5.0%

[Heavy Metal] ≤10PPM

[Storage] Store in cool & dry area, keep away from the direct light and heat.

[Package] Packed in paper-drums and two plastic-bags inside.

[What is Rhodiola Rosea]

Rhodiola Rosea (also known as Arctic root or golden root) is a member of the family Crassulaceae, a family of plants native to the arctic regions of Eastern Siberia. Rhodiola rosea is widely distributed  in Arctic and mountainous regions throughout Europe and Asia. It grows at altitudes of 11,000 to 18,000 feet above sea level.

There are numerous animal and test tube studies showing that rhodiola has both a stimulating and a sedating effect on the central nervous system; enhance physical endurance; improves thyroid, thymus, and adrenal function; protects the nervous system, heart and liver; and has antioxidant and anticancer properties.

[Function]

1 Enhancing immunity and delaying aging;

2 Resisting radiation and tumor;

3 Regulating nervous system and metabolism, effectively limiting melancholy feeling and mood, and promoting mental status;

4 Protecting cardiovascular, dilating coronary artery,preventing coronary arteriosclerosis and arrhythmia.

https://SuccesswithTroyandDana.com
Sugar Blocker | F21 All Natural Sugar Blocker | Ludaxx Sugar Blocker Works!

F21 has been specifically formulated as an All Natural Sugar Blocker™ to help limit your blood sugar absorption. For every tablet of F21 you can block up to 10 grams of sugar (sucrose). Additionally, our patent pending formula not only helps promote weight loss, it benefits the digestive system by allowing the blocked sucrose to support beneficial probiotic bacteria while the polysaccharide (PSK) boosts your immune system response.

F21 Ingredients:
L-Arabinose, Coriolus Versicolor Polysacchride, Konjac-Mannan, Magnesium Stearate
Mint flavor: Menthol and Natural Colors.

Dr Rudi’s Story
“As a medical weight loss specialist, I am so excited to have access to products that supports what I have been telling my patients for years at a cost that is affordable. One of the biggest problems my clients face is convincing them to make changes in their lifestyle before they start seeing success, now with F21 and the other Ludaxx products, changes can begin to happen even before their lifestyle has changed.”

What is F21?
F21 is a delicious tasting tablet that is half the sweetness of sugar which helps block sugar absorption into the bloodstream as well as supporting beneficial probiotics within your digestive system! It is also an antioxidant that will boost your immune system response…

Cameron’s Story
“I decided to join Ludaxx after I started seeing my friends and family that were using F21 begin to experience better health including weight loss. Even though the need to lose weight is not an issue that I am facing, knowing that every gram of F21 can block up to twenty grams of Sucrose makes me feel less worried that weight will ever be an issue for me.”

F21 has been formulated to help limit your blood sugar absorption and control further weight gain. For every gram of L-Arabinose, F21 blocks up to 20 grams of sugar (sucrose) by inhibiting the enzyme called sucrase. Our digestive system will also receive the benefits of F21 to feed probiotic bacteria. With our powerful polysaccharides (PSK) to support the immune system response and protect against further free radical damage, we present you the product of the future. *

* These statements have not been evaluated by the Food and Drug Administration (FDA). This product is not intended to diagnose, prevent, mitigate, treat or cure disease.

Ingredients
L-Arabinose, Coriolus versicolor polysaccharide,

Other Ingredients
Konjac-Mannan, Magnesium Stearate

Help us share the power of the F21 with others…

Thanks, Troy & Dana
904-591-5413

https://successwithtroyanddana.com

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“Equine Polysaccharide Storage Myopathy” is an inheritable glycogen storage disease of horses that causes exertional rhabdomyolysis. It is most commonly associated with heavy horse breeds and the American Quarter Horse. While incurable, PSSM can be managed with appropriate diet and exercise. There are currently 2 subtypes, known as Type 1 PSSM and Type 2 PSSM.

Glycogen is a molecular polymer of glucose used to store energy, and is important for maintaining glucose hemostasis in the blood, as well as for providing energy for skeletal muscle and cardiac muscle contraction. Molecules of glucose are linked into linear chains by α-1,4-glycosidic bonds. Additionally, branches of glucose are formed off of the chain by α-1,6-glycosidic bonds. 2 molecules of glucose are joined into an α-1,4-glycosidic bonds by an enzyme known as glycogen synthase. This bond may be broken by amylase when the body wishes to break down glycogen into glucose for energy. Glycogen branching enzyme is responsible for the required α-1,6-glycosidic bonds needed to start a branch off of these linear chains.

Any disruption to this system results in a glycogen storage disease. There are currently 3 subcategories of glycogen storage diseases in horses: Type 1 Polysaccharide Storage Myopathy, Glycogen Branching Enzyme Deficiency , and Type 2 Polysaccharide Storage Myopathy.

Type 1 PSSM is caused by an autosomal dominant genetic mutation known as GSY1. This mutation causes an up-regulation of glycogen synthase, and high levels of glycogen synthase relative to glycogen branching enzyme . This altered ratio of glycogen synthase to GBE results in glycogen molecules with long chains and few branches, making these molecules somewhat amylase resistant. The GSY1 mutation is associated with altered glucose metabolism , as well as accumulation of high levels of glycogen and abnormal polysaccharide in the muscles of the horse. Additionally, some horses have been shown to have insulin sensitivity, which improves glucose uptake by muscle cells and contributes to excessive glycogen storage that is already elevated secondary to the GSY1 mutation.

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